Human aggression across the lifespan: genetic propensities and environmental moderators

Citation

Tuvblad, C. & Baker, L. A. (2011). Human aggression across the lifespan: genetic propensities and environmental moderators. Adv Genet. vol. 75 pp. 171-214 , PMCID: PMC3696520

Abstract

This chapter reviews the recent evidence of genetic and environmental influences on human aggression. Findings from a large selection of the twin and adoption studies that have investigated the genetic and environmental architecture of aggressive behavior are summarized. These studies together show that about half (50%) of the variance in aggressive behavior is explained by genetic influences in both males and females, with the remaining 50% of the variance being explained by environmental factors not shared by family members. Form of aggression (reactive, proactive, direct/physical, indirect/relational), method of assessment (laboratory observation, self-report, ratings by parents and teachers), and age of the subjects-all seem to be significant moderators of the magnitude of genetic and environmental influences on aggressive behavior. Neither study design (twin vs. sibling adoption design) nor sex (male vs. female) seems to impact the magnitude of the genetic and environmental influences on aggression. There is also some evidence of gene-environment interaction (G x E) from both twin/adoption studies and molecular genetic studies. Various measures of family adversity and social disadvantage have been found to moderate genetic influences on aggressive behavior. Findings from these G x E studies suggest that not all individuals will be affected to the same degree by experiences and exposures, and that genetic predispositions may have different effects depending on the environment.

URL

http://dx.doi.org/10.1016/b978-0-12-380858-5.00007-1

Keyword(s)

Adoption

Notes

Tuvblad, Catherine

Reference Type

Journal Article

Journal Title

Adv Genet

Author(s)

Tuvblad, C.
Baker, L. A.

Year Published

2011

Volume Number

75

Pages

171-214

Edition

2011/11/15

DOI

10.1016/b978-0-12-380858-5.00007-1

PMCID

PMC3696520

NIHMSID

Nihms378907

Reference ID

4565